scottbushey wrote:Thinking..........
I don't quite understand something:
If Pauling says that by taking High dose VC and suplementing that with Proline and Lysine, the high cholesterol should not be the issue. The high dose VC therapy should counteract the inflamation; The cholesterol should lose it's sticky property and not adher to the arterial wall. The root canaled teeth issue would be moot.......I'm confused.
I do not think Pauling's position was that "high cholesterol would not be the issue" (in the sense that it may still rise) but you might infer that elevated cholesterol would not be as dangerous as long as you are taking Lp(a) binding inhibitors. So yes, if I were in your position, I would be taking C to bowel tolerance, and lysine (and probably proline, but lets think about it below.)
Dr. Levy's opinion is based on his clinical experience with patients who did not reverse their symptoms until their root canals/amalgams were removed, but we do not know the dosages of ascorbate these patients were taking. It is possible they were taking much lower vitamin C dosages than we (Pauling) advise. I'll ask Dr. Levy. Your cholesterol may stay elevated until the dental toxins are removed, but the issue is what then happens to your cardiovascular system? (And it seems logical that the elevated cholesterol is acting in your behalf - helping your body eliminate the toxins.)
The central issue is the strength and stability of the arterial wall. Your objective is to keep your arteries strong - despite the probability that vitamin C in your blood stream may be dissipated before it can be utilized for the production of collagen (in the arterial wall.) Levy in his book discusses the many apparent alternative means the body seems to use to stabilize the arterial wall (and ground substance), and the associated "thickening" that results from this process. This is one of the reasons he recommends Chrondroitin, etc.
As Pauling once said regarding all this, "What do we know?" It is logical that taking more vitamin C is prudent, whether the ascorbate is being used to help you eliminate toxins, or is being utilized to produce collagen.
If there are other means (e.g. IP6) of detoxifying root canals, i.e., that can help with the probable toxic load, then that would, in theory, free up more ascorbate for the role of collagen production. We do know that a mere 10 mg of vitamin C is enough to prevent frank scurvy. So you are correct, there seems to be a disconnect. Given how lipsonal C is thought to operate, low-energy entry into the cells, it is conceivable that this form would be best and at least one Lypo-C packet should be included in your regimen.
The issue of Lp(a) binding inhibitors is not so clear cut. Lysine seems to have two faces. There are lysine strands in the collagen matrix, so lysine is at least a component of collagen/elastin, but lysine in the blood can also inactivate the sticky Lp(a) molecules. In theory, the elevated Lp(a) is providing a service if your arteries are growing weaker, missing collagen. If we knew that your Lp(a) was quite elevated, then more lysine/proline (in theory) would be helpful to avoid "overshooting" the patch. Your elevated blood pressure argues for this (arterial narrowing).
I think it boils down to the danger from the toxins is a secondary effect of reducing collagen production by reducing available vitamin C. Given that Lp(a) apparently rises when C is unavailable, forgetting the complication of the toxic load, then your Lp(a) reading may be the best indicator and may suggest the best future course of action. If your Lp(a) were low, for example, it would say to me that your current regimen is working.