Moderator: ofonorow
Say I am an individual wanting you to analyze and score my retinal images,
a) Wll you?
YES The first exam is free in the UK and anywhere else World wide. They pay the Optometrist, not me ever.
b) how much will it cost me?
NOTHING
c) what kind of image can you analyze
any from a 45 degree camera that is in good focus and exposure
d) should I ask my doctor for anything specific?
No NOTHING AT ALL
e) How long will it take?
From receipt of the image depends on workload 10 to 30 mins to report
f) Do I get a free 700+ Secrets.. (kidding)
After signing up for the 3 yr contract YES.. AND I already give your book to my registrants!
g) how much for an optional nutritional analysis?
Once you are registered it's part of the service. NOT UNTIL. Takes too long.
Hi Owen,
Thank you very much indeed for taking the trouble to send me these images
which are particularly interesting and I hope they are not yours.
Both images are of excellent quality and are fairly easy to work with.
They would have been better still for quicker and easier evaluation had the fixation been the same for both.
But I am pleased to have been able to do what you asked without annotating the critical points of interest in the images which require closer attention.
However:
I must clarify some issues:
1.) The service that I provide is a personal and private one.
2.) I accept images from Optometrists for evaluation on the basis that the image rights belong to the Institute and are not for publication by others without permission.
3.) The Institute is not giving free training and instruction on the Internet.
4.) The Optometrist is at liberty to see the comments I make on the images if the individual wishes to show him them, but Optometrists have little or no experience and they have been taught wrongly about retinal atherosclerosis.
They therefore fail to understand the importance of the detail. It is simply not yet part of their training and, far worse, they will continue to be instructed wrongly in Optometry schools in the UK and the USA until I rewrite the section on retinal arteriolar and venular atheroma in the text book.
5.) I am not going to do that until my first paper is forced into a Reed Elsevier
Optometry journal, all of which are defending the false medical shibboleth (dogma)
that arterial disease is irreversible whilst refusing to publish Pauling-Rath theory on which Nutritional Preventive CardioRetinometry is founded, and without knowledge of which - the phenomenon would not have been discovered.
6.) Again, whilst the Optometrist sending the images can be asked by the patient to comment at any stage, none in the USA is, as yet, competent to do so.
7.) A second opinion on the images should be available from Dr. Vera Riches MD via
the Institute who will probably make a charge of £30 or more for each paired evaluation
8.) I am reasonably confident that Dr. Riches will always be happy to give an opinion on two sets of images having spent 3 full years studying the subject and acquiring the skill to evaluate the images. She expects to be the first to earn the DCardioRet, probably this year.
OPINION:
1. This patient is particularly vulnerable to glaucoma. I am not saying he/she has it.
2. The atherosclerosis in the image of 2009 is Grade 2.5+. That is not good at all. It is worse than average
3.) The image of two years later shows progression of atherogenesis which is now grade 3.0
I do not often see images at this stage. Ordinarily I would ask for a signed 'consent to treat' and waiver of liability. Medicine has little to offer except statins and their record is not good.
A recent review stated that they had proved better in secondary prevention than in primary prevention. The public appears to fail to understand that what is being said here is that one has to experience a first heart attack to perhaps receive a benefit in reduced risk of a second.
4.) The images could be from a male or female. They suggest that regression of the atherosclerosis might be accomplished before kidney or other serious damage occurs, but a thrombosis is inevitable if the condition continues to deteriorate and - in any case - I have seen thrombosis occur before this stage of arterial disease has been reached. I would not be surprised if this person has already had a coronary thrombosis.
In the USA I would write to the doctor (there is no cooperation at all in the UK) and advise continuous and vigorous Pauling therapy, suggested with some vessels almost completely blocked
I do not want to say more.
Feel free to publish this but I do not wish it to become expected of me.
If you wish to edit it, please allow me to see your text before publication.
Yours sincerely,
Sydney Bush
2. The atherosclerosis in the image of 2009 is Grade 2.5+. That is not good at all. It is worse than average
3.) The image of two years later shows progression of atherogenesis which is now grade 3.0
I know Dr. Bush has reported seeing positive improvement in less than a month on his nutritional program on the "soft" atheromas - which features sodium ascorbate, vitamin E, etc.
Phlebotomy helps a lot
Could the VC be responsible for the high Ferritin readings, as VC increase the uptake of Iron?
Owen,
I saw the lab report, and everything now makes sense to me. That is an alarm level of iron in your body, as iron is really the final common denominator for a large amount, perhaps most, of the oxidative stress generated in the body, in this case in the arterial wall. The advice is still the same: phlebotomy, far infrared sauna, and IP6. Although traditional medicine would have you believe otherwise, these three measures can get the iron level down fairly quickly, perhaps as much as 100 ng/cc every 6-month period or so. I partake of all three measures myself, and my once-very-elevated ferritin is now at 28 ng/cc. If a large amount of research directly addressing is desired, then forget it. That is why it is not well-appreciated. But all the evidence is available in the literature, if you look long enough and hard enough.
This is why I repeat and emphasize: ferritin level, until a even better iron storage test comes about, should be a part of ALL routine blood testing, right along with CBC and biochemistry panel. But most docs don't even know what a ferritin level is or why it is important.
So, to recap,
1. Phlebotomy, 6 units annually (some center will take two units at one time, every 4 months)
2. Far infrared sauna, minimum of 3 times weekly, daily if possible; I stay in 30 minutes at 125 to 140 degrees, and I usually put out in excess of 700 cc sweat
3. IP6, 2 to 4 grams on an empty stomach
The sauna will waste magnesium and electrolytes; taking a broad spectrum vegetable/fruit powder ("greens") will keep things in balance.
Best regards,
Dr. Levy
Hello Owen,
I was determined to end my participation on this thread, since so much, if not all, of the advice seems to have been ignored. Regardless, I am more concerned about someone's health here rather than opinions on my advice. Gofanu is way off base. It's very interesting that he dismissed all the points about the relationship of elevated iron in an artery to atherosclerosis, while adding that he doesn't intend on wasting his time becoming educated on the ferritin blood test.
When you have rapidly progressive atherosclerosis and dental toxins have been largely eliminated (especially the root canals), an elevated ferritin level of 495 is quite astronomical for someone who does not have hemochromatosis, and it is almost certainly a major player if not the primary player in that particular case of rapid atherosclerosis evolution. Fenton chemistry, which generally requires iron, is the main reason oxidative stress is present and continues to be present in the arterial wall.
While nothing is for sure in biology and medicine, I believe the progression of atherosclerosis would markedly slow, if not stop completely, and even start reversing with the supplements being taken, if the ferritin level can be dropped below 30. Much slowing should occur if it can be dropped below 150.
Marathon athletes routinely exercise themselves to iron deficiency anemia due to the iron lost in the sweat. That recommendation was not some kind of idle comment. Same with the IP6.
OK, that's it. No more comments on this thread. I wish jknosplr and cobraman the best.
Best regards,
Dr. Levy
No salt restrictions use Sea salt onlyI'm willing to bet you are on salt restriction
Not to my knowledge with the two quarts of water plus taken daily with the VC/lysine/proline/protocol, plus other liquids during course of the day.I'll hazard a guess that you are chronically dehydrated,
I was determined to end my participation on this thread, since so much, if not all, of the advice seems to have been ignored. Regardless, I am more concerned about someone's health here rather than opinions on my advice. Gofanu is way off base. It's very interesting that he dismissed all the points about the relationship of elevated iron in an artery to atherosclerosis, while adding that he doesn't intend on wasting his time becoming educated on the ferritin blood test.
While nothing is for sure in biology and medicine, I believe the progression of atherosclerosis would markedly slow, if not stop completely, and even start reversing with the supplements being taken, if the ferritin level can be dropped below 30. Much slowing should occur if it can be dropped below 150.
FERRITIN:
Ferritin is the most important measure of iron. Iron in the body is mostly in hemoglobin and myoglobin. However, iron is also used by every single cell in the body as part of many enzymes. Many of the enzymes which participate in the citric acid cycle to generate ATP - the basic energy storage unit in the body - in mitochondria have iron in their structure. Ferritin gives one an idea of how much iron is available to the rest of the body's cells for metabolic purposes.
Without iron, cells are significantly impaired in metabolic activity. They can't make enough ATP to do their activities. Thus, optimizing hormone, neurotransmitters and other signals doesn't work very well since they are only signals. They are signals to trigger cellular activities. But these activities cannot be done without ATP.
An optimum iron level as measured by Ferritin in men is about 150. In women, it is about 100-120. These are mid-range values. A ferritin of 75, in one study, was found to be the lower end of normal for senior citizens. They can be even develop iron-deficiency anemia at that level of iron.
Excessive iron is dangerous. It is highly oxidizing. It is destructive to tissues - causing cell death in the testes, ovaries, thyroid gland, liver, brain, etc. In testing Ferritin, I have surprisingly found a large number of patients, who have been treatment resistant, to have hemochromocytosis - a disease of excessive iron storage.
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