majkinetor wrote:Some additions:
The info is OK, but without the dose, its meaningless.
ofonorow wrote:I note that vitamin C is in almost every category (as it should be!). If there is a category without vitamin C, then perhaps that category needs to be reexamined!
By the way, not to abuse your crib topic, but this is really what we are saying. And what Dr. Levy has deduced, along with Pauling/Rath.
I agree with you about correct Vitamin C dosage, when people are having heart attacks after getting the correct dosage and asking what went wrong? couldn't it be easy to overlook an ingredient that may possibly make the difference in staving off another MI? Enhancements in the protocol evolve surely?
Magnesium is now recognized as a first-line medicine for the treatment of heart attacks. , A study published in The Lancet, for example, reported the effects of a double-blind, randomized, placebo-controlled study in 2,316 patients with suspected myocardial infarction. The dose of magnesium was high (about 8.7 grams given intravenously over a 24 hour period), but the results were remarkable: magnesium reduced cardiovascular mortality by 25 percent. The author/s conclusion:
"Intravenous magnesium sulfate is a simple, safe, and widely applicable treatment. Its efficacy in reducing early mortality of myocardial infarction is comparable to, but independent of, that of thrombolytic or antiplatelet therapy."
These findings have been confirmed and reconfirmed in many clinics and laboratories. Teo and colleagues, for example, in an analysis of seven clinical studies, concluded that magnesium (in doses of 5-10 grams by intravenous injection) reduced the odds of death by an astounding 55%.
(Too bad these papers/abstracts are hard to find using PUBMED.GOV - e.g. http://www.ncbi.nlm.nih.gov/pubmed/8504519 )
Studies of magnesium have revealed it to be Nature's "calcium-channel blocker" ; unlike its drug counterparts, however, magnesium has no toxic side-effects. Another important effect of supplemental magnesium is its ability to mitigate the cardiotoxic effects of catecholamines. Prielipp and associates, for example, published results of a clinical trial in which magnesium (10 mg per kg body weight per hour, or approximately 700 mg per hour for an average adult) attenuated the cardiotoxic effects of epinephrine in 17 bypass patients. Interestingly, the drug captopril - an angiotensin- converting enzyme (ACE) inhibitor - has been demonstrated to work by raising intracellular magnesium.
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