Bacterial Infections
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Ascorbate in massive doses markedly augments the effects of
antibiotics and broadens their spectrum greatly. Ascorbate also prevents
most, if not all, allergic reactions to necessary antibiotics. Ascorbate
seems to manage these feats by neutralizing the massive amounts of free radicals
generated in the infected tissues when mitochondria are damaged. These
free radicals then neutralize all of the vitamin C in the affected tissues.
This state of acute induced scurvy turns off
the white cells. Their phagocytosis requires a least a little vitamin C.
When electrons are driven into the involved tissues with
large doses of oral ascorbic acid or, if necessary
massive doses of sodium ascorbate intravenously, the white cells come out
fighting mad and destroy the infection. (Remember the reason for the
massive doses of ascorbate is that we are mostly throwing away the vitamin C for
the electrons carried.) Eliminating the free radicals eliminates the
inflammation, increasing circulation to the area of infection, which allows the
antibiotic to reach the affected tissues better. While at the same time
the elimination of the oxidative redox potential reduces
the affinity of antibodies for their antigen and this prevents the allergic
reaction to the antibiotic. I have not seen a Staphylococcus aureus
infection resistant to massive doses of ascorbate plus a first generation
antibiotic such as the Keflex mentioned in the article.
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This case is especially tragic because massive doses of
ascorbate seem to neutralize the toxin of beta hemolytic strep group A because
it cures scarlet fever and rheumatic fever in a few hours. These are very
interesting because they are cured so easily and rapidly. We have to
protect the profits of the drug companies however. If the public were to
discover how easily diseases involving free radicals were cured, it would be a
financial disaster for the drug companies. To hell with the patients.
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Most of these patients die free radical deaths. Regardless of the
bacteria involved, free radicals are released largely because of damage to
mitochondria. There are also various toxins involved, most of which
are free radicals or at least some toxin which ascorbate in adequate amounts
will neutralize. In any event all these patients, in addition to the
standard antibiotic and supportive care, should be given
massive doses of intravenous sodium ascorbate.
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These massive doses of ascorbate will reverse the acute induced scurvy
induced by the free radicals of the infection in the affected parts.
One of the problems in serious infections is that the massive amounts of
free radicals destroy all the vitamin C in the area of the infection.
The white cells need some ascorbate to perform phagocytosis. The first
step of the respiratory burst of the white cells is the reduction of
molecular oxygen to superoxide. This reduction power ordinarily comes
from NADPH but this can be destroyed in the oxidative redox potential in the
infected areas. The massive doses of ascorbate by forcing a reducing
redox potential onto the infected tissues prevent this shutdown of the
respiratory burst.
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Additionally, these massive doses of ascorbate prevent allergic
reactions to the necessary antibiotics. Antibodies are turned on in an
oxidative redox potential and since adequate ascorbate will maintain a
reducing redox potential even in the infected tissues, the antibodies are
not turned on.
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One might worry that if the antibodies are not turned on, the infection
will persist. Actually, the antibody system is a backup system for the
white cells and if the white cells keep fighting the antibodies are not
necessary. In the area where all the free radicals are neutralized and
a reducing redox potential is maintained, there is not inflammation
(redness, swelling, pain). However, if not adequate amounts of
ascorbate are delivered to the affected tissues, they will fail to prevent
an oxidative redox potential and the antibodies will be turned on in that
localized area. This is why it is better to turn off the antibodies
with ascorbate rather than cortisone, methotrexate, cyclosporin, etc.
because they suppress the immune system generally. Ascorbate turns off
the antibodies only by eliminating the free radicals that turn on the
antibodies but augments the respiratory burst of the white cells.
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Besides that, massive doses of ascorbate prevent the free radical
inflammatory cascade that damage cells beyond the actually infected areas.
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I know, I know that if the public learns of all this it will cut down on
the necessity for drugs and be a financial disaster for the drug industry
but come on gang what about the lives of these people.
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The favorite trick to deny this ascorbate effect of vitamin C is to use
inadequate doses. Do not listen to the idiots who research one, two,
four, etc gram doses. Depending upon the severity of the disease, it
takes 60, 100, 500 grams of ascorbate to accomplish this ascorbate effect.
This is a matter of chemistry, not medicine; the amount of ascorbate
necessary to eliminate all the free radicals must be used. Lesser
amounts do not work and provide ample publishable results that say that
ascorbate does not work. Adequate, massive amounts of ascorbate
suddenly eliminate all the free radicals of various diseases and that is
what it takes to make ascorbate work. We are not talking about the
usual vitamin functions of vitamin C. Studies of the blood levels of
vitamin C in well people are ridiculous. What we are interested in is
the tissue levels of ascorbate in the depths of the tissues involved in the
infection.
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The problem with these deaths is probably they are all
free radical deaths. It is not so
much that the bug is in every way more powerful than other bugs, it is that
they have developed the ability to not be killed by antibiotics. All
these bugs damage mitochondria and those damaged mitochondria release many
free radicals. These free radicals suppress the immune systems ability
to kill the bugs. These free radicals can easily be neutralized by
massive doses of sodium ascorbate given intravenously.
Then the immune system can easily eliminate the bug.
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Whenever a seriously infected case presents, massive doses of
intravenous sodium ascorbate should be started. This will prevent the
free radical deaths regardless of the cause of the infection.
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Among other things, massive doses of ascorbate increase the spectrum of
activity of necessary antibiotics.
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If the infection is not very severe bowel
tolerance ascorbic acid orally should be started immediately. A
throat culture or culture of wherever should be taken. If by history
or if the culture is positive for a bacteria an appropriate antibiotic
should be started (if the patient is not obviously getting well on the
ascorbate alone.) In severe cases massive doses of
intravenous sodium ascorbate should be started
(along with the appropriate antibiotic determined by culture) and kept going
until the patient is mostly well, then ascorbic acid by mouth can be
substituted until completely well. If the bacteria cultures a resistant
bacteria, a first or second generation antibiotic should be given anyway
because the ascorbate will broadly increase the spectrum of activity of the
antibiotic and will prevent allergic reactions to the antibiotic.
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When these severely ill patients present, they should be started on
massive doses of intravenous sodium ascorbate.
These massive doses, not the small doses written about in the literature,
will prevent the free radical deaths.